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FOXC1 Monoklonaler Antikörper

FOXC1 Monoklonal Antikörper für WB, Indirect ELISA

Wirt / Isotyp

Maus / IgG1

Getestete Reaktivität

human

Anwendung

WB, Indirect ELISA

Konjugation

Unkonjugiert

CloneNo.

1F4E11

Kat-Nr. : 66568-1-PBS

Synonyme

FREAC 3, Forkhead-related transcription factor 3, Forkhead-related protein FKHL7, FKHL7, ARA

Filter:
  • Western Blot (WB) analysis of various lysates using FOXC1 Monoclonal antibody (66568-1-Ig)

    WB analysis using 66568-1-Ig (same clone as 66568-1-PBS)

    Various cells were subjected to SDS PAGE followed by western blot with 66568-1-Ig (FOXC1 antibody) at dilution of 1:10000 incubated at room temperature for 1.5 hours. This data was developed using the same antibody clone with 66568-1-PBS in a different storage buffer formulation.

  • Western Blot (WB) analysis of HEK-293 cells using FOXC1 Monoclonal antibody (66568-1-Ig)

    WB analysis of HEK-293 using 66568-1-Ig (same clone as 66568-1-PBS)

    WB result of FOXC1 antibody (66568-1-Ig; 1:10000; incubated at room temperature for 1.5 hours) with sh-Control and sh-FOXC1 transfected HEK-293 cells. This data was developed using the same antibody clone with 66568-1-PBS in a different storage buffer formulation.

Geprüfte Anwendungen

Produktinformation

66568-1-PBS bindet in WB, Indirect ELISA FOXC1 und zeigt Reaktivität mit human

Getestete Reaktivität human
Wirt / Isotyp Maus / IgG1
Klonalität Monoklonal
Typ Antikörper
Immunogen Peptid
Vollständiger Name forkhead box C1
Berechnetes Molekulargewicht 57 kDa
Beobachtetes Molekulargewicht 70-75 kDa
GenBank-ZugangsnummerNM_001453
Gene symbol FOXC1
Gene ID (NCBI) 2296
Konjugation Unkonjugiert
Form Liquid
Reinigungsmethode Protein-G-Reinigung
Lagerungspuffer PBS only
LagerungsbedingungenStore at -80°C. 20ul Größen enthalten 0,1% BSA.

Hintergrundinformationen

FOXC1, also named as FKHL7 and FREAC3, binding of FREAC-3 and FREAC-4 to their cognate sites results in bending of the DNA at an angle of 80-90 degrees. Defects in FOXC1 are the cause of Axenfeld-Rieger syndrome type 3 (RIEG3). Defects in FOXC1 are the cause of iridogoniodysgenesis anomaly (IGDA). Defects in FOXC1 are a cause of Peters anomaly.