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KCNJ9 Polyklonaler Antikörper

KCNJ9 Polyklonal Antikörper für WB, Indirect ELISA

Wirt / Isotyp

Kaninchen / IgG

Getestete Reaktivität

human, Maus, Ratte

Anwendung

WB, Indirect ELISA

Konjugation

Unkonjugiert

Kat-Nr. : 31742-1-PBS

Synonyme

G protein-activated inward rectifier potassium channel 3, GIRK 3, GIRK3, GIRK-3, KIR3.3

Filter:
  • Western Blot (WB) analysis of various lysates using KCNJ9 Polyclonal antibody (31742-1-AP)

    WB analysis using 31742-1-AP (same clone as 31742-1-PBS)

    Various lysates were subjected to SDS PAGE followed by western blot with 31742-1-AP (KCNJ9 antibody) at dilution of 1:2000 incubated at room temperature for 1.5 hours. This data was developed using the same antibody clone with 31742-1-PBS in a different storage buffer formulation.

Geprüfte Anwendungen

Produktinformation

31742-1-PBS bindet in WB, Indirect ELISA KCNJ9 und zeigt Reaktivität mit human, Maus, Ratten

Getestete Reaktivität human, Maus, Ratte
Wirt / Isotyp Kaninchen / IgG
Klonalität Polyklonal
Typ Antikörper
Immunogen KCNJ9 fusion protein Ag35873
Vollständiger Name potassium inwardly-rectifying channel, subfamily J, member 9
Beobachtetes Molekulargewicht38-44 kDa
GenBank-ZugangsnummerBC167777
Gene symbol KCNJ9
Gene ID (NCBI) 3765
Konjugation Unkonjugiert
Form Liquid
Reinigungsmethode Antigen-Affinitätsreinigung
Lagerungspuffer PBS only
LagerungsbedingungenStore at -80°C. 20ul Größen enthalten 0,1% BSA.

Hintergrundinformationen

KCNJ9, also known as GIRK3 (G protein-activated inwardly rectifying potassium channel 3), is a crucial subunit that forms inwardly rectifying potassium channels. These channels are primarily activated by inhibitory G proteins (Gi/o) downstream of various G protein-coupled receptors (GPCRs), such as those for neurotransmitters like GABA, acetylcholine, and adenosine. Upon activation, KCNJ9 facilitates potassium efflux, leading to membrane hyperpolarization and a reduction in neuronal excitability. It is widely expressed in the brain, where it plays a key role in regulating synaptic transmission and neuronal circuits, influencing processes ranging from pain perception to reward and motivation.