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OPA3 Monoklonaler Antikörper

OPA3 Monoklonal Antikörper für WB, Indirect ELISA

Wirt / Isotyp

Maus / IgG1

Getestete Reaktivität

human

Anwendung

WB, Indirect ELISA

Konjugation

Unkonjugiert

CloneNo.

5A3F9

Kat-Nr. : 68241-1-PBS

Synonyme

FLJ22187, FLJ25932, MGA3, OPA3, Optic atrophy 3 protein

Filter:
  • Western Blot (WB) analysis of various lysates using OPA3 Monoclonal antibody (68241-1-Ig)

    WB analysis using 68241-1-Ig (same clone as 68241-1-PBS)

    Various lysates were subjected to SDS PAGE followed by western blot with 68241-1-Ig (OPA3 antibody) at dilution of 1:3000 incubated at room temperature for 1.5 hours. This data was developed using the same antibody clone with 68241-1-PBS in a different storage buffer formulation.

  • Western Blot (WB) analysis of various lysates using OPA3 Monoclonal antibody (68241-1-Ig)

    WB analysis using 68241-1-Ig (same clone as 68241-1-PBS)

    Various lysates were subjected to SDS PAGE followed by western blot with 68241-1-Ig (OPA3 antibody) at dilution of 1:3000 incubated at room temperature for 1.5 hours. This data was developed using the same antibody clone with 68241-1-PBS in a different storage buffer formulation.

Geprüfte Anwendungen

Produktinformation

68241-1-PBS bindet in WB, Indirect ELISA OPA3 und zeigt Reaktivität mit human

Getestete Reaktivität human
Wirt / Isotyp Maus / IgG1
Klonalität Monoklonal
Typ Antikörper
Immunogen OPA3 fusion protein Ag8173
Vollständiger Name optic atrophy 3 (autosomal recessive, with chorea and spastic paraplegia)
Berechnetes Molekulargewicht 179 aa, 20 kDa
Beobachtetes Molekulargewicht 20 kDa
GenBank-ZugangsnummerBC005059
Gene symbol OPA3
Gene ID (NCBI) 80207
Konjugation Unkonjugiert
Form Liquid
Reinigungsmethode Protein-G-Reinigung
Lagerungspuffer PBS only
LagerungsbedingungenStore at -80°C. 20ul Größen enthalten 0,1% BSA.

Hintergrundinformationen

The OPA3 cDNA encodes a deduced 179-amino acid protein. Northern blot analysis demonstrated a primary transcript of approximately 5.0 kb that was ubiquitously expressed, most prominently in skeletal muscle and kidney. Mutations in this gene have been shown to result in 3-methylglutaconic aciduria type III and autosomal dominant optic atrophy and cataract.