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STMN3 Monoklonaler Antikörper

STMN3 Monoklonal Antikörper für WB, Indirect ELISA

Wirt / Isotyp

Maus / IgG2b

Getestete Reaktivität

human

Anwendung

WB, Indirect ELISA

Konjugation

Unkonjugiert

CloneNo.

1F12B1

Kat-Nr. : 66152-1-PBS

Synonyme

1F12B1, SCG10-like protein, SCLIP, Stathmin 3, stathmin like 3

Filter:
  • Western Blot (WB) analysis of human brain tissue using STMN3 Monoclonal antibody (66152-1-Ig)

    WB analysis of human brain using 66152-1-Ig (same clone as 66152-1-PBS)

    human brain tissue were subjected to SDS PAGE followed by western blot with 66152-1-Ig (STMN3 antibody) at dilution of 1:1000 incubated at room temperature for 1.5 hours. This data was developed using the same antibody clone with 66152-1-PBS in a different storage buffer formulation.

Geprüfte Anwendungen

Produktinformation

66152-1-PBS bindet in WB, Indirect ELISA STMN3 und zeigt Reaktivität mit human

Getestete Reaktivität human
Wirt / Isotyp Maus / IgG2b
Klonalität Monoklonal
Typ Antikörper
Immunogen STMN3 fusion protein Ag18844
Vollständiger Name stathmin-like 3
Berechnetes Molekulargewicht 21 kDa
Beobachtetes Molekulargewicht 21 kDa
GenBank-ZugangsnummerBC009381
Gene symbol STMN3
Gene ID (NCBI) 50861
Konjugation Unkonjugiert
Form Liquid
Reinigungsmethode Protein-A-Reinigung
Lagerungspuffer PBS only
LagerungsbedingungenStore at -80°C. 20ul Größen enthalten 0,1% BSA.

Hintergrundinformationen

Stathmin family phosphoproteins participate in the control of microtubule dynamics and have been proposed to be involved in the control of neuronal differentiation. Neuron specific Stathmin-3 (STMN3) is a novel STAT3 (signal transducer and activator of transcription 3) interacting protein. STAT3 is a key contributor to cancer cell migration and invasion. STMN3 exhibits microtubule-destabilizing activity, which is antagonized by STAT3. SCG10 is a widely studied and recognized neuronal differentiation marker, STMN3 is a SCG10-like protein (SCLIP) and is present from the earliest stages of hippocampal neuron differentiation in culture at vesicle-like structures following dynamic microtubules. RNAi mediated inhibition of this gene resulted in increased axonal branching.