Anticorps Monoclonal anti-FOXC1

• Phare
• Validé par KD/KO

FOXC1 Monoclonal Antibody for WB, Indirect ELISA

Hôte / Isotype

Mouse / IgG1

Réactivité testée

Humain

Applications

WB, Indirect ELISA

Conjugaison

Non conjugué

CloneNo.

1F4E11

N° de cat : 66568-1-PBS

Synonymes

FREAC 3, Forkhead-related transcription factor 3, Forkhead-related protein FKHL7, FKHL7, ARA

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Galerie de données de validation

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  WB analysis using 66568-1-Ig (same clone as 66568-1-PBS)

  Various cells were subjected to SDS PAGE followed by western blot with 66568-1-Ig (FOXC1 antibody) at dilution of 1:10000 incubated at room temperature for 1.5 hours. This data was developed using the same antibody clone with 66568-1-PBS in a different storage buffer formulation.

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  WB analysis of HEK-293 using 66568-1-Ig (same clone as 66568-1-PBS)

  WB result of FOXC1 antibody (66568-1-Ig; 1:10000; incubated at room temperature for 1.5 hours) with sh-Control and sh-FOXC1 transfected HEK-293 cells. This data was developed using the same antibody clone with 66568-1-PBS in a different storage buffer formulation.

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Informations sur le produit

66568-1-PBS cible FOXC1 dans les applications de WB, Indirect ELISA et montre une réactivité avec des échantillons Humain

• Réactivité: Humain
• Hôte / Isotype: Mouse / IgG1
• Clonalité: Monoclonal
• Type: Anticorps
• Immunogène: Peptide
• Nom complet: forkhead box C1
• Masse moléculaire calculée: 57 kDa
• Poids moléculaire observé: 70-75 kDa
• Numéro d’acquisition GenBank: NM_001453
• Symbole du gène: FOXC1
• Identification du gène (NCBI): 2296
• Conjugaison: Non conjugué
• Forme: Liquide
• Méthode de purification: Purification par protéine G
• Tampon de stockage: PBS only
• Conditions de stockage: Store at -80°C. 20ul contiennent 0,1% de BSA.

Informations générales

FOXC1, also named as FKHL7 and FREAC3, binding of FREAC-3 and FREAC-4 to their cognate sites results in bending of the DNA at an angle of 80-90 degrees. Defects in FOXC1 are the cause of Axenfeld-Rieger syndrome type 3 (RIEG3). Defects in FOXC1 are the cause of iridogoniodysgenesis anomaly (IGDA). Defects in FOXC1 are a cause of Peters anomaly.

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