Anticorps Monoclonal anti-OPA3

OPA3 Monoclonal Antibody for WB, Indirect ELISA

Hôte / Isotype

Mouse / IgG1

Réactivité testée

Humain

Applications

WB, Indirect ELISA

Conjugaison

Non conjugué

CloneNo.

5A3F9

N° de cat : 68241-1-PBS

Synonymes

FLJ22187, FLJ25932, MGA3, OPA3, Optic atrophy 3 protein

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Galerie de données de validation

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  WB analysis using 68241-1-Ig (same clone as 68241-1-PBS)

  Various lysates were subjected to SDS PAGE followed by western blot with 68241-1-Ig (OPA3 antibody) at dilution of 1:3000 incubated at room temperature for 1.5 hours. This data was developed using the same antibody clone with 68241-1-PBS in a different storage buffer formulation.

• 

  WB analysis using 68241-1-Ig (same clone as 68241-1-PBS)

  Various lysates were subjected to SDS PAGE followed by western blot with 68241-1-Ig (OPA3 antibody) at dilution of 1:3000 incubated at room temperature for 1.5 hours. This data was developed using the same antibody clone with 68241-1-PBS in a different storage buffer formulation.

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Informations sur le produit

68241-1-PBS cible OPA3 dans les applications de WB, Indirect ELISA et montre une réactivité avec des échantillons Humain

• Réactivité: Humain
• Hôte / Isotype: Mouse / IgG1
• Clonalité: Monoclonal
• Type: Anticorps
• Immunogène: OPA3 Protéine recombinante Ag8173
• Nom complet: optic atrophy 3 (autosomal recessive, with chorea and spastic paraplegia)
• Masse moléculaire calculée: 179 aa, 20 kDa
• Poids moléculaire observé: 20 kDa
• Numéro d’acquisition GenBank: BC005059
• Symbole du gène: OPA3
• Identification du gène (NCBI): 80207
• Conjugaison: Non conjugué
• Forme: Liquide
• Méthode de purification: Purification par protéine G
• Tampon de stockage: PBS only
• Conditions de stockage: Store at -80°C. 20ul contiennent 0,1% de BSA.

Informations générales

The OPA3 cDNA encodes a deduced 179-amino acid protein. Northern blot analysis demonstrated a primary transcript of approximately 5.0 kb that was ubiquitously expressed, most prominently in skeletal muscle and kidney. Mutations in this gene have been shown to result in 3-methylglutaconic aciduria type III and autosomal dominant optic atrophy and cataract.

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