TGFBI/BIGH3 Recombinant antibody, PBS Only

TGFBI/BIGH3 Uni-rAbTM Recombinant Antibody for WB, Indirect ELISA
Cat No. 80805-4-PBS
Clone No.243108F7

Host / Isotype

Rabbit / IgG

Reactivity

human

Applications

WB, Indirect ELISA

TGFBI, TGFBI / BIGH3, Beta ig h3, Beta ig-h3, BIGH3

Formulation:  PBS Only
PBS and Azide
PBS Only
Conjugate:  Unconjugated
Size/Concentration: 

-/ -

Freight/Packing: -

Quantity

Please visit your regions distributor:


Product Information

80805-4-PBS targets TGFBI/BIGH3 in WB, Indirect ELISA applications and shows reactivity with human samples.

Tested Reactivity human
Host / Isotype Rabbit / IgG
Class Recombinant
Type Antibody
Immunogen TGFBI/BIGH3 fusion protein Eg1233 Predict reactive species
Full Name transforming growth factor, beta-induced, 68kDa
Calculated Molecular Weight75kDa
Observed Molecular Weight68 kDa
GenBank Accession NumberNM_000358.3
Gene Symbol TGFBI
Gene ID (NCBI) 7045
ENSEMBL Gene IDENSG00000120708
Conjugate Unconjugated
Form Liquid
Purification MethodProtein A purification
UNIPROT IDQ15582
Storage Buffer PBS only, pH 7.3.
Storage ConditionsStore at -80°C.

Background Information

TGFBI, also named as BIGH3, Kerato-epithelin and RGD-CAP, binds to type I, II, and IV collagens. TGFBI is an adhesion protein which may play an important role in cell-collagen interactions. In cartilage, it may be involved in endochondral bone formation. TGFBI is an extracellular matrix adaptor protein, it has been reported to be differentially expressed in transformed tissues. TGFBI is a predictive factor of the response to chemotherapy, and suggest the use of TGFBI-derived peptides as possible therapeutic adjuvants for the enhancement of responses to chemotherapy.(PMID:20509890) Defects in TGFBI are the cause of epithelial basement membrane corneal dystrophy (EBMD). Defects in TGFBI are the cause of corneal dystrophy Groenouw type 1 (CDGG1). Defects in TGFBI are the cause of corneal dystrophy lattice type 1 (CDL1). Defects in TGFBI are a cause of corneal dystrophy Thiel-Behnke type (CDTB). Defects in TGFBI are the cause of Reis-Buecklers corneal dystrophy (CDRB). Defects in TGFBI are the cause of lattice corneal dystrophy type 3A (CDL3A). Defects in TGFBI are the cause of Avellino corneal dystrophy (ACD).

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