SKIL Polyclonal antibody
SKIL Polyclonal Antibody for IP, WB, ELISA
Host / Isotype
Rabbit / IgG
Reactivity
human
Applications
WB, IP, CoIP, ELISA
Conjugate
Unconjugated
Cat no : 19218-1-AP
Synonyms
Validation Data Gallery
Tested Applications
Positive WB detected in | A431 cells, HepG2 cells, human skeletal muscle tissue |
Positive IP detected in | HepG2 cells |
Recommended dilution
Application | Dilution |
---|---|
Western Blot (WB) | WB : 1:500-1:2000 |
Immunoprecipitation (IP) | IP : 0.5-4.0 ug for IP and 1:500-1:1000 for WB |
Sample-dependent, check data in validation data gallery |
Published Applications
WB | See 2 publications below |
CoIP | See 2 publications below |
Product Information
The immunogen of 19218-1-AP is SKIL Fusion Protein expressed in E. coli.
Tested Reactivity | human |
Cited Reactivity | human |
Host / Isotype | Rabbit / IgG |
Class | Polyclonal |
Type | Antibody |
Immunogen | SKIL fusion protein Ag5282 |
Full Name | SKI-like oncogene |
Calculated molecular weight | 77 kDa |
Observed molecular weight | 77 kDa |
GenBank accession number | BC059386 |
Gene symbol | SKIL |
Gene ID (NCBI) | 6498 |
Conjugate | Unconjugated |
Form | Liquid |
Purification Method | Antigen affinity purification |
Storage Buffer | PBS with 0.02% sodium azide and 50% glycerol pH 7.3. |
Storage Conditions | Store at -20°C. Stable for one year after shipment. Aliquoting is unnecessary for -20oC storage. |
Protocols
Product Specific Protocols | |
---|---|
WB protocol for SKIL antibody 19218-1-AP | Download protocol |
IP protocol for SKIL antibody 19218-1-AP | Download protocol |
Standard Protocols | |
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Click here to view our Standard Protocols |
Publications
Species | Application | Title |
---|---|---|
Biochim Biophys Acta De novo GLI3 mutation in esophageal atresia: Reproducing the phenotypic spectrum of Gli3 defects in murine models. | ||
Cell Death Dis SKIL facilitates tumorigenesis and immune escape of NSCLC via upregulating TAZ/autophagy axis. | ||
J Cell Mol Med MDM2 is implicated in high-glucose-induced podocyte mitotic catastrophe via Notch1 signalling. | ||
J Neurosci The Transcriptional Regulator SnoN Promotes the Proliferation of Cerebellar Granule Neuron Precursors in the Postnatal Mouse Brain. | ||
Cell Death Differ PIAS1 and TIF1γ collaborate to promote SnoN SUMOylation and suppression of epithelial-mesenchymal transition. |