Phospho-SYN1 (Ser9) Recombinant monoclonal antibody

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  WB analysis of mouse brain using 87293-2-RR

  mouse brain tissue were subjected to SDS PAGE followed by western blot with 87293-2-RR (Phospho-SYN1 (Ser9) antibody) blocked with BSA only, Phospho-SYN1 (Ser9) peptide or non-Phospho peptide at dilution of 1:40000 incubated at room temperature for 1.5 hours.

Phospho-SYN1 (Ser9) Uni-rAb^® Recombinant Antibody for WB, ELISA

Cat No. 87293-2-RR

Clone No.252630C2

Host / Isotype

Rabbit / IgG

Reactivity

human, mouse

Applications

WB, ELISA

Synonyms

Phospho SYN1 (Ser9), Phospho Synapsin (Ser9), Phospho-SYN1, Phospho-Synapsin, Phospho-Synapsin (Ser9)

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Tested Applications

• Positive WB detected in: mouse brain tissue

Recommended dilution

• Western Blot (WB): WB : 1:5000-1:50000

It is recommended that this reagent should be titrated in each testing system to obtain optimal results.

Sample-dependent, Check data in validation data gallery.

Product Information

87293-2-RR targets Phospho-SYN1 (Ser9) in WB, ELISA applications and shows reactivity with human, mouse samples.

• Tested Reactivity: human, mouse
• Host / Isotype: Rabbit / IgG
• Class: Recombinant
• Type: Antibody
• Immunogen:

  Peptide

• Full Name: synapsin I
• Calculated Molecular Weight: 74 kDa
• Observed Molecular Weight: 77 kDa
• GenBank Accession Number: NM_006950
• Gene Symbol: SYN1
• Gene ID (NCBI): 6853
• Conjugate: Unconjugated
• Form: Liquid
• Purification Method: Protein A purification
• UNIPROT ID: P17600
• Storage Buffer: PBS with 0.02% sodium azide and 50% glycerol, pH 7.3.
• Storage Conditions: Store at -20°C. Stable for one year after shipment. Aliquoting is unnecessary for -20^oC storage. 20ul sizes contain 0.1% BSA.

Background Information

SYN1, also named Brain protein 4.1, belongs to the synapsin family. Phospho-SYN1 (Ser9) is a phosphorylated form of Synapsin 1 (SYN1) at serine residue 9, which plays a significant role in the regulation of synaptic vesicle dynamics. The phosphorylation of Syn1 at this site is induced by action potentiation, leading to the disassociation of Syn1 with its interacting proteins and the subsequent release of synaptic vesicles (SVs).