FOXG1 Recombinant antibody, PBS Only

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  WB analysis using 85930-1-RR (same clone as 85930-1-PBS)

  Various lysates were subjected to SDS PAGE followed by western blot with 85930-1-RR (FOXG1 antibody) at dilution of 1:10000 incubated at room temperature for 1.5 hours. This data was developed using the same antibody clone with 85930-1-PBS in a different storage buffer formulation.

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  IF Staining of C6 using 85930-1-RR (same clone as 85930-1-PBS)

  Immunofluorescent analysis of (4% PFA) fixed C6 cells using FOXG1 antibody (85930-1-RR, Clone: 250144E8 ) at dilution of 1:800 and CoraLite®488-Conjugated Goat Anti-Rabbit IgG(H+L) (SA00013-2), CL594-Phalloidin (red). This data was developed using the same antibody clone with 85930-1-PBS in a different storage buffer formulation.

FOXG1 Uni-rAb^TM Recombinant Antibody for WB, IF/ICC, Indirect ELISA

Cat No. 85930-1-PBS

Clone No.250144E8

Host / Isotype

Rabbit / IgG

Reactivity

human, mouse, rat

Applications

WB, IF/ICC, Indirect ELISA

Synonyms

FOXG 1, BF-1, BF-2, forkhead box G1, Forkhead box protein G1

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Product Information

85930-1-PBS targets FOXG1 in WB, IF/ICC, Indirect ELISA applications and shows reactivity with human, mouse, rat samples.

• Tested Reactivity: human, mouse, rat
• Host / Isotype: Rabbit / IgG
• Class: Recombinant
• Type: Antibody
• Immunogen: FOXG1 fusion protein Ag17169
• Full Name: forkhead box G1
• Calculated Molecular Weight: 489 aa, 52 kDa
• Observed Molecular Weight: 60 kDa
• GenBank Accession Number: BC035020
• Gene Symbol: FOXG1
• Gene ID (NCBI): 2290
• Conjugate: Unconjugated
• Form: Liquid
• Purification Method: Protein A purification
• UNIPROT ID: P55316
• Storage Buffer: PBS only, pH 7.3.
• Storage Conditions: Store at -80°C.

Background Information

Forkhead Box G1 (FOXG1) is a member of the Forkhead family of genes with non-redundant roles in brain development, where alteration of this gene's expression significantly affects the formation and function of the mammalian cerebral cortex. FOXG1 haploinsufficiency in humans is associated with prominent differences in brain size and impaired intellectual development noticeable in early childhood, while homozygous mutations are typically fatal.